compensatory hypertrophy

Protein synthesis and muscle fiber composition in compensatory hypertrophy of rat plantaris muscle

骨骼肌代偿性肥大时蛋白质合成速度与肌纤维类型的变化

Q & a on sex effect of intermittent starvation on the estrous cycles and compensatory hypertrophy of ovary in rats

间歇饥饿对大鼠动情周期及卵巢代偿性肥大的影响

Objective : To investigate the animal model of guinea pig chronic heart failure and compensatory hypertrophy .

目的：探讨豚鼠慢性充血性心力衰竭（CHF）及致心肌肥厚的动物模型制作方法。

CONCLUSION : Myocardial apoptosis is one of the possible mechanisms for the development of compensatory hypertrophy to heart failure .

结论：心肌细胞凋亡是代偿性心肌肥厚发展为心力衰竭的可能机制之一。

Objective : To investigate the compensatory hypertrophy in kidney transplants after surgery and its related factors .

目的：探讨肾移植术后移植肾的代偿性增生情况。

Transient Compensatory Hypertrophy and Limited Regeneration of the Surgically Removed Rat Ventral Prostate

手术摘除大鼠腹侧前列腺后暂时代偿性肥大与限制性再生

Clinical Observations of Compensatory Hypertrophy in Kidney Transplants

移植肾代偿性增生的临床观察

In the healthy rats , the affected segment was atrophic and the remaining liver underwent compensatory hypertrophy .

正常大鼠SPVE后局部肝段萎缩，其余肝叶代偿性增大。

CONCLUSIONS : It is feasible , safe and effective that PVE induce compensatory hypertrophy of the remnant liver in clinical use .

结论：门静脉栓塞诱导肝叶代偿性增生在临床上的应用是可行的、安全的、有效的。

Objective To evaluate the usefulness and safety of portal vein branch embolization inducing compensatory hypertrophy of the remnant liver by absolute ethanol .

目的研究无水酒精犬门静脉分支栓塞诱导肝叶代偿增生的有效性与安全性。

In numerous studies of mechanism which myocardial compensatory hypertrophy develops to heart failure , the phenomenon of apoptosis has been observed , which has been got more attention .

在心肌代偿性肥厚发展为心力衰竭机制的众多研究中，细胞凋亡的现象被人们发现，并日益受到重视。

In the second , there were myocardial apoptosis , perivascular fibrosis and apoptosis in compensatory hypertrophy heart caused by pressure overloading during cardiac remodeling .

心肌间质纤维化、血管周围纤维化和心肌细胞凋亡参与压力超负荷高血压大鼠代偿性心肌肥厚阶段心脏重构的病理过程。

Objective To evaluate the clinical feasibility , safety , and effectiveness of percutaneous right portal vein embolization ( PVE ) inducing compensatory hypertrophy of the left liver .

目的评估门静脉右支栓塞诱导左肝叶代偿性增生临床应用的可行性、安全性、有效性。

Kidney stereopsis analysis showed the improvement of kidney compensatory hypertrophy and glomerulosclerosis in groups of SMI combination with dialysis was better than that of single dialysis group ;

肾脏体镜视学分析显示，参麦配合腹透对肾脏代偿性肥大及肾小球硬化的改善作用显著好于单纯腹透组，且以腹透加参麦腹腔内使用效果最好。

Conclusion : The animal model of guinea pig chronic heart failure and compensatory hypertrophy may be formed by constricting the ascending aortas of guinea pigs for 6 weeks .

结论：升主动脉缩窄6周，豚鼠CHF及致心肌肥厚的动物模型基本形成。

Results : In comparison between the control group of 3 months age , with that of 15,18 and 21-24 months age , the adrenal cortex of aged rats compensatory hypertrophy with significant increase of lipid droplet , lipofuscin and mitochondria in reticular zone ( P < 0.01 ) .

结果：3月龄与15、18和21～24月龄C组比较，老年大鼠肾上腺皮质代偿性增生，网状带胞浆内的线粒体脂滴和脂褐素面积增加（P<0.01）。

Alterations of EGF activities in the early stage of compensatory renal hypertrophy following unilateral nephrectomy in rats

肾切除术后留存肾代偿早期过程中EGF活性的实验研究

Treatment of compensatory turbinate hypertrophy with radiofrequency ablation

低温等离子射频消融术治疗代偿性下鼻甲肥大

Post infarcted remodeling , which including infarct expansion , compensatory reactive hypertrophy of the noninfarcted zone , and subsequently global ventricular dilatation , profoundly affects prognosis for survival .

心肌梗塞后心室重塑包括梗塞区膨展、非梗塞区反应性肥厚、晚期进行性球形心室扩张，对心室功能和预后都有着深远的影响。

Conclusion Vitamin E can suppress fibrosis of remnant kidney in 5 / 6 nephrectomy induced CRF rats , and does not affect directly the compensatory glomerular hypertrophy and glomerular capillary proliferation .

结论维生素E能显著抑制5/6肾切除所致慢性肾衰残肾组织的纤维化，对其代偿性肾小球肥大和毛细血管增生无明显直接影响。

Results In group B , accompanied with compensatory glomerular hypertrophy and glomerular capillary proliferation , there were obvious glomerular sclerosis , filtration membrane thickening , interstitial fibrosis , and renal hydroxyproline content increase .

结果大鼠行5/6肾切除术后，在残存肾小球代偿性肥大、肾小球毛细血管代偿性增生的同时，肾小球显著硬化，滤过膜显著增厚，肾间质显著纤维化，肾组织羟脯氨酸含量显著增高；

Although pathological hypertrophy of the myocardium is temporarily compensatory , prolonged hypertrophy may result in ventricular arrhythmias , heart failure , and subsequent cardiovascular mortality .

虽然心肌细胞最初的生理性肥大是一种代偿机制，但是持续长久的心肌肥厚可能导致心律失常、心力衰竭甚至猝死。

According to the report , there are two types of renal compensatory growth process including hypertrophy which means the enlargement of the cell and regeneration which means the increase of the cell quantity that both affected by animal age .

据文献报道，肾代偿由两种形式&肥大和再生完成。肥大是指肾组织细胞的体积增加，再生是指代偿时肾组织细胞数目增多。

Objective To study the role of connective tissue growth factor ( CTGF ) and vascular endothelial growth factor ( VEGF ) in the early stage of compensatory renal growth , and to explore the mechanisms of renal compensatory hypertrophy on rat kidney following unilateral nephrectomy .

目的观察一侧肾切除大鼠术后留存肾早期结缔组织生长因子（CTGF）及血管内皮生长因子（VEGF）表达的改变，探讨一侧肾切除大鼠留存肾的代偿及肥大的机制。